Metabolic Bone Disease
Nutritional secondary hyperparathyroidism
Auch bekannt als: Nutritional Secondary Hyperparathyroidism, NSHP, Fibrous Osteodystrophy, Osteomalacia, Rickets
Kurz gesagt
Metabolic Bone Disease (MBD), or nutritional secondary hyperparathyroidism, is a common and serious condition in captive reptiles and turtles. Caused by a lack of dietary calcium, improper calcium-to-phosphorus ratios, or insufficient UVB light, it leads to weak, deformed bones and muscle tremors. Learn how to recognize the signs, how veterinarians diagnose and treat it, and how proper husbandry can prevent this debilitating disease.

TL;DR. Verhinderbare Störung bei Reptilien und Schildkröten durch Calcium- oder UVB-Mangel: weiche Knochen, Deformitäten und Tremor.
UVB-Licht ist essenziell für Vitamin-D3-Synthese und Calciumaufnahme.
Was ist das?
Metabolic bone disease in captive reptiles/turtles is usually nutritional secondary hyperparathyroidism: insufficient calcium or UVB leads to soft bones, fibrous reinforcement of limbs/jaws, and muscle tremors. Calcium is pulled from bone to keep blood calcium safe.
Ursachen und Risikofaktoren
Husbandry errors: inadequate UVB (290-315 nm), low dietary calcium, poor Ca:P ratio (aim near 2:1), cold enclosures. Fast-growing species are highest risk.
Anzeichen
- Rubber jaw; soft/warped shell in chelonians (cardinal)
- Swollen firm limbs, pathological fractures, lethargy, anorexia, tremors (common)
- Paresis/paralysis from spinal deformity (occasional)

Verkürzter Kiefer und geschwollene Beine sind fortgeschrittene Klassiker.
Diagnose
Husbandry review. Radiographs (gold): demineralized ghost bones, hidden fractures. Ionized calcium; biochemistry for kidney and phosphorus.

Röntgen zeigt entkalkte Knochen und verborgene Frakturen.
Behandlung
Oral or injectable calcium salts; phosphate binders if phosphorus high; cautious calcitriol after calcium stabilizes. Fix UVB, heat gradient, diet.
Prognose
Excellent if early before severe deformity. Permanent shape changes may remain. Poor with paralysis or severe kidney disease.
Vorbeugung
Linear UVB (replace every 6-12 months), calcium (± D3 indoors), gut-load insects, Ca-rich greens, correct thermal gradient.
Wann den Tierarzt rufen
Emergency: continuous tremors/seizures, inability to lift body, soft jaw/shell, visible fractures, extreme lethargy or prolonged anorexia.
Quellen
- Mader's Reptile and Amphibian Medicine and Surgery, 3rd Edition, pages 315-321, 811-814.
- BSAVA Manual of Reptiles, 3rd Edition, pages 102-105, 210-212.
My highlights & notes
Anzeichen & Symptome
Diagnose
- RadiographyGoldstandard
- Plasma Biochemistry Panel
- Plasma Ionized Calcium
Behandlungsansätze
Behandlungen müssen von einem approbierten Tierarzt verordnet werden. Konkrete Dosierungen werden absichtlich nicht angezeigt.
Häufige Fragen
What is Metabolic Bone Disease?
Metabolic Bone Disease (MBD), or nutritional secondary hyperparathyroidism, is a common and serious condition in captive reptiles and turtles. Caused by a lack of dietary calcium, improper calcium-to-phosphorus ratios, or insufficient UVB light, it leads to weak, deformed bones and muscle tremors. Learn how to recognize the signs, how veterinarians diagnose and treat it, and how proper husbandry can prevent this debilitating disease.
What are the symptoms of Metabolic Bone Disease?
Soft, deformed carapace or plastron、Soft, pliable mandible or maxilla (rubber jaw)、Loss of appetite、Lethargy and weakness、Muscle tremors or twitching、Pathological fractures、Swollen, firm limbs、Paresis or paralysis
How is Metabolic Bone Disease diagnosed?
Radiography、Plasma Biochemistry Panel、Plasma Ionized Calcium
How is Metabolic Bone Disease treated?
Behandlungen müssen von einem approbierten Tierarzt verordnet werden. Konkrete Dosierungen werden absichtlich nicht angezeigt.
Dieser Artikel dient der allgemeinen Information und ersetzt keinen tierärztlichen Rat. Bei Krankheit bitte einen Tierarzt aufsuchen.
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